To truly control neurological diseases, we must solve both why they begin and why they progress.
While I pursue the molecular mechanisms of onset in patient-derived cells, I believe that clues to progression and deterioration lie hidden in patients’ clinical course seen in everyday practice.
Cells are where mechanisms are decoded; the clinic is a treasury of hints about progression—yet research that cleanly connects the two does not yet exist.
As one of the few researchers holding both a basic and a clinical perspective, I want to bridge this gap.
Carrying causes decoded in cells toward the prevention of clinical progression, and returning clinical insights to the cell, I aim to achieve cause-based prevention of onset and progression in neurological diseases—both pursued in equal measure.